Angiotensin II/en: Unterschied zwischen den Versionen
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| − | The substance formed by [[ACE]] from [[angiotensin I]]. Angiotensin II is the most potent [[ | + | The substance formed by [[ACE/en|ACE]] from [[Angiotensin_I/en|angiotensin I]]. Angiotensin II is the most potent [[Vasokonstriktion,_vasokonstriktorisch/en|vasoconstrictive]]<nowiki />effective and thus blood pressure-increasing substance in the body. A. also stimulates the release of [[Aldosteron/en|aldosterone]] from the adrenal cortex and increases the activity of the [[Sympathisch/en|sympathetic]]. A. is the most important substance for medium-term blood pressure regulation. The main task of angiotensin II is to increase blood pressure on three levels: |
| − | 1. constriction of the [[arterioles]] above the sympathetic nervous system | + | 1. constriction of the [[Arteriolen/en|arterioles]] above the sympathetic nervous system |
| − | 2. | + | 2. reduced excretion of water in the kidneys by hormones ([[ADH,_antidiuretisches_Hormon,_Vasopressin/en|ADH]] and aldosterone) |
| − | + | Third, thirst. | |
| − | + | These mechanisms are designed to increase blood pressure. | |
[https://de.wikipedia.org/wiki/Angiotensin_II Angiotensin II in Wikipedia]<sub> ([https://de.wikipedia.org/wiki/Wikipedia:Lizenzbestimmungen_Commons_Attribution-ShareAlike_3.0_Unported Wikipedia CC-by-sa-3.0])</sub> | [https://de.wikipedia.org/wiki/Angiotensin_II Angiotensin II in Wikipedia]<sub> ([https://de.wikipedia.org/wiki/Wikipedia:Lizenzbestimmungen_Commons_Attribution-ShareAlike_3.0_Unported Wikipedia CC-by-sa-3.0])</sub> | ||
Aktuelle Version vom 22. Mai 2019, 00:23 Uhr
The substance formed by ACE from angiotensin I. Angiotensin II is the most potent vasoconstrictiveeffective and thus blood pressure-increasing substance in the body. A. also stimulates the release of aldosterone from the adrenal cortex and increases the activity of the sympathetic. A. is the most important substance for medium-term blood pressure regulation. The main task of angiotensin II is to increase blood pressure on three levels:
1. constriction of the arterioles above the sympathetic nervous system
2. reduced excretion of water in the kidneys by hormones (ADH and aldosterone)
Third, thirst.
These mechanisms are designed to increase blood pressure.