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Its biosynthesis is mediated by the [[adrenocorticotropic hormone|adrenocorticotropic hormone]] (ACTH) from the anterior pituitary. Aldosterone stimulates Na<sup>+</sup>-reabsorption and K<sup>+</sup>-secretion in the salt-transporting cells of the kidney and leads to retention of Na<sup>+</sup> and increased excretion of K<sup>+</sup> throughout the body. | Its biosynthesis is mediated by the [[adrenocorticotropic hormone|adrenocorticotropic hormone]] (ACTH) from the anterior pituitary. Aldosterone stimulates Na<sup>+</sup>-reabsorption and K<sup>+</sup>-secretion in the salt-transporting cells of the kidney and leads to retention of Na<sup>+</sup> and increased excretion of K<sup>+</sup> throughout the body. | ||
− | + | The secondary result is water retention, and thus an increase in [[extracellular volume|extracellular volume]]. | |
[https://de.wikipedia.org/wiki/Aldosteron Aldosteron in Wikipedia] <sub>([https://de.wikipedia.org/wiki/Wikipedia:Lizenzbestimmungen_Commons_Attribution-ShareAlike_3.0_Unported Wikipedia CC-by-sa-3.0])</sub> | [https://de.wikipedia.org/wiki/Aldosteron Aldosteron in Wikipedia] <sub>([https://de.wikipedia.org/wiki/Wikipedia:Lizenzbestimmungen_Commons_Attribution-ShareAlike_3.0_Unported Wikipedia CC-by-sa-3.0])</sub> |
Most important adrenalbark hormone for the regulation of salt metabolism (mineralocorticoid).
Its biosynthesis is mediated by the adrenocorticotropic hormone (ACTH) from the anterior pituitary. Aldosterone stimulates Na+-reabsorption and K+-secretion in the salt-transporting cells of the kidney and leads to retention of Na+ and increased excretion of K+ throughout the body.
The secondary result is water retention, and thus an increase in extracellular volume.