Acetylcholin/en: Unterschied zwischen den Versionen
| Zeile 6: | Zeile 6: | ||
Clinically, descendants of the arrow poison of the Indians, the curare, are used for muscle relaxation during anaesthesia. They competitively block the acetylcholine receptors at the motor endplates. This will force all muscles to relax. | Clinically, descendants of the arrow poison of the Indians, the curare, are used for muscle relaxation during anaesthesia. They competitively block the acetylcholine receptors at the motor endplates. This will force all muscles to relax. | ||
| − | + | Some insecticides irreversibly inhibit the [[Enzym/en|enzyme]] that breaks down acetylcholine. The acetylcholine can then no longer be broken down at the motor end plates, which results in an increase in the neurotransmitter concentration. This can lead to a fatal muscle spasm. | |
https://en.wikipedia.org/wiki/Acetylcholine <sub>([https://en.wikipedia.org/wiki/Wikipedia:Text_of_Creative_Commons_Attribution-ShareAlike_3.0_Unported_License])</sub> | https://en.wikipedia.org/wiki/Acetylcholine <sub>([https://en.wikipedia.org/wiki/Wikipedia:Text_of_Creative_Commons_Attribution-ShareAlike_3.0_Unported_License])</sub> | ||
Version vom 12. Juli 2019, 20:16 Uhr
Neurotransmitter of the interneuronal and neuroeffective signal transmission to acetylcholine receptors. Essential transmitter in the vegetative nervous system, especially in the parasympathetic nervous system.
Acetylcholine is on the one hand a neurotransmitter of the vegetative ganglia and on the other hand a neurotransmitter for the transmission of the nerve impulse from the efferent motoric neurons to the muscle. It has a stimulating effect on the acetylcholine receptors of the downstream structures.
Clinically, descendants of the arrow poison of the Indians, the curare, are used for muscle relaxation during anaesthesia. They competitively block the acetylcholine receptors at the motor endplates. This will force all muscles to relax.
Some insecticides irreversibly inhibit the enzyme that breaks down acetylcholine. The acetylcholine can then no longer be broken down at the motor end plates, which results in an increase in the neurotransmitter concentration. This can lead to a fatal muscle spasm.